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THE REMARKABLE BENEFITS OF COCONUT OIL

By Ray Peat

This is a slightly modified version of Ray Peat’s article which can be found at http://www.efn.org/~raypeat/
He does some fine paintings too!

I have already discussed the many toxic effects of the unsaturated oils, and I have frequently mentioned that coconut oil doesn’t have those toxic effects,
though it does contain a small amount of the unsaturated oils.

Many people have asked me to write something on coconut oil. I thought I might write a small book on it, but I realize that there are no suitable channels for distributing such a book — if the seed-oil industry can eliminate major corporate food products that have used coconut oil for a hundred years, they certainly
have the power to prevent dealers from selling a book that would affect their market more seriously.

For the present, I will just outline some of the virtues of coconut oil.

The unsaturated oils in some cooked foods become rancid in just a few hours, even at refrigerator temperatures, and are responsible for the stale taste
of leftover foods. (Eating slightly stale food isn’t particularly harmful, since the same oils, even when eaten absolutely fresh, will oxidize at a much
higher rate once they are in the body, where they are heated and thoroughly mixed with an abundance of oxygen.)

Coconut oil that has been kept at room temperature for a year has been tested for rancidity, and showed no evidence of it. Since we would expect the small
percentage of unsaturated oils naturally contained in coconut oil to become rancid, it seems that the other (saturated) oils have an antioxidative effect:
I suspect that the dilution keeps the unstable unsaturated fat molecules spatially separated from each other, so they can’t interact in the destructive chain
reactions that occur in other oils.

To interrupt chain-reactions of oxidation is one of the functions of antioxidants, and it is possible that a sufficient quantity of coconut oil in the body has this
function. It is well established that dietary coconut oil reduces our need for vitamin E, but I think its antioxidant role is more general than that, and that it has both direct and indirect antioxidant activities.

Coconut oil is unusually rich in short and medium chain fatty acids.
Shorter chain length allows fatty acids to be metabolized without use of the carnitine transport system. Mildronate protects cells against
stress partly by opposing the action of carnitine, and comparative studies showed that added carnitine had the opposite effect, promoting the
oxidation of unsaturated fats during stress, and increasing oxidative damage to cells.

I suspect that a degree of saturation of the oxidative apparatus by short-chain fatty acids has a similar effect — that is, that these very soluble and mobile short-chain saturated fats have priority for oxidation, because they don’t require carnitine transport into the mitochondrion, and that this will tend to inhibit oxidation of the unstable, peroxidizable unsaturated fatty acids.

When Albert Schweitzer operated his clinic in tropical Africa, he said it was many years before he saw any cases of cancer, and he believed that the appearance of cancer was caused by the change to the European type of diet. In the l920s, German researchers showed that mice on a fat-free diet were practically free of cancer.
Since then, many studies have demonstrated a very close association between consumption of unsaturated oils and the incidence of cancer.

Heart damage is easily produced in animals by feeding them linoleic acid; this “essential” fatty acid turned out to be the heart toxin in rape-seed oil. (talker says is now called’canola’ oil) The addition of saturated fat to the experimental heart-toxic oil-rich diet protects against the damage to heart cells.
Immunosuppression was observed in patients who were being “nourished” by intravenous emulsions of “essential fatty acids,” and as a result coconut oil is used as the basis for intravenous fat feeding, except in organ-transplant patients.
For those patients, emulsions of unsaturated oils are used specifically for their immunosuppressive effects.

General aging, and especially aging of the brain, is increasingly seen as being closely associated with lipid peroxidation. Several years ago I met an old couple, who were only a few years apart in age, but the wife looked many years younger than her doddering old husband. She was from the Philippines, and she remarked that she always had to cook two meals at the same time, because her husband couldn’t adapt to her traditional food. Three times every day, she still prepared her
food in coconut oil. Her apparent youth increased my interest in the effects of coconut oil.

In the l960s, Hartroft and Porta gave an elegant argument for decreasing the ratio of unsaturated oil to saturated oil in the diet (and thus in the tissues). They showed that the “age pigment” is produced in proportion to the ratio of oxidants to antioxidants, multiplied by the ratio of unsaturated oils to saturated oils.
More recently, a variety of studies have demonstrated that ultraviolet light induces peroxidation in unsaturated fats, but not saturated fats, and that this occurs in the skin as well as in the lab. Rabbit experiments, and studies of humans, showed that the amount of unsaturated oil in the diet strongly affects the
rate at which aged, wrinkled skin develops. The unsaturated fat in the skin is a major target for the aging and carcinogenic effects of ultraviolet light, though
not necessarily the only one. In the l940s, farmers attempted to use cheap coconut oil for fattening their animals, but they found that it made them lean, active and hungry. For a few years, an antithyroid drug was found to make the livestock get fat while eating less food, but then it was found to be a strong carcinogen, and it also probably produced hypothyroidism in the people who ate the meat.

By the late l940s, it was found that the same antithyroid effect, causing animals to get fat without eating much food, could be achieved by using soy beans and corn as feed. Later, an animal experiment fed diets that were low or high in total fat, and in different groups the fat was provided by pure coconut oil, or a pure unsaturated oil, or by various mixtures of the two oils. At the end of their lives, the animals’ obesity increased directly in proportion to the ratio of unsaturated oil to coconut oil in their diet, and was not related to the total amount of fat they had consumed. That is, animals which ate just a little pure unsaturated oil were fat, and animals which ate a lot of coconut oil were lean.

G. W. Crile and his wife found that the metabolic rate of people in Yucatan, where coconut is a staple food, averaged 25% higher than that of people in the United States. In a hot climate, the adaptive tendency is to have a lower metabolic rate, so it is clear that some factor is more than offsetting this expected effect of high environmental temperatures. The people there are lean, and recently it has been observed that the women there have none of the symptoms we commonly associate with the menopause.

By l950, then, it was established that unsaturated fats suppress the metabolic rate, apparently creating hypothyroidism. Over the next few decades, the exact mechanisms of that metabolic damage were studied. Unsaturated fats damage the mitochondria, partly by suppressing the reparatory enzyme, and partly by causing generalized oxidative damage. The more unsaturated the oils are, the more specifically they suppress tissue response to thyroid hormone, and transport of the
hormone on the thyroid transport protein.

Plants evolved a variety of toxins designed to protect themselves from “predators,” such as grazing animals. Seeds contain a variety of toxins, that seem to be specific for mammalian enzymes, and the seed oils themselves function to block protein digestive enzymes in the stomach. The thyroid hormone is formed in the gland by the action of a protein digestive enzyme, and the unsaturated oils also inhibit that enzyme. Similar protein digestive enzymes involved in clot removal and immune function appear to be similarly inhibited by these oils.

Just as metabolism is “activated” by consumption of coconut oil, which prevents the inhibiting effect of unsaturated oils, other inhibited processes, such as clot removal and immune function, will probably tend to be restored by continuing use of coconut oil.

Brain tissue is very rich in complex forms of fats.
The experiment (around 1978) in which pregnant mice were given diets containing either coconut oil or unsaturated oil showed that brain development was superior in the young mice whose mothers ate coconut oil. Because coconut oil supports thyroid function, and thyroid governs brain development, including myelination, the result
might simply reflect the difference between normal and hypothyroid individuals.
However, in 1980, experimenters demonstrated that young rats fed milk containing soy oil incorporated the oil directly into their brain cells, and had structurally abnormal brain cells as a result. Lipid oxidation occurs during seizures, and antioxidants such as vitamin E have some anti-seizure activity. Currently,
lipid oxidation is being found to be involved in the nerve cell degeneration of Alzheimer’s disease.
Various fractions of coconut oil are coming into use as “drugs,” meaning that they are advertised as treatments for diseases. Butyric acid is used to treat cancer, lauric and myristic acids to treat virus infections, and mixtures of medium-chain fats are sold for weight loss.

Purification undoubtedly increases certain effects, and results in profitable products, but in the absence of more precise knowledge, I think the whole natural product, used as a regular food, is the best way to protect health.
The shorter-chain fatty acids have strong, unpleasant odors; for a couple of days after I ate a small amount of a medium-chain triglyceride mixture, my skin oil emitted a rank, goaty smell. Some people don’t seem to have that reaction, and the benefits might outweigh the stink, but these things just haven’t been in use long enough to know whether they are safe.

Treating any complex natural product as the drug industry does, as a raw material to be fractionated in the search for “drug” products, is risky, because the relevant knowledge isn’t sought in the search for an association between a single chemical and a single disease.
While the toxic unsaturated paint-stock oils, especially safflower, soy, corn and linseed (flaxseed) oils, have been sold to the public precisely for their drug effects, all of their claimed benefits were false.
When people become interested in coconut oil as a “health food,” the huge seed-oil industry — operating through their shills — are going to attack it as an “unproved drug.”

While components of coconut oil have been found to have remarkable physiological effects (as antihistamines, antiinfectives/antiseptics, promoters of immunity, glucocorticoid antagonist, nontoxic anticancer agents, for example).
The cholesterol-lowering fiasco for a long time centered on the ability of unsaturated oils to slightly lower serum cholesterol. For years, the mechanism of that action wasn’t known, which should have suggested caution. Now, it seems that the effect is just one more toxic action, in which the liver defensively retains its cholesterol, rather than releasing it into the blood.

Large scale human studies have provided overwhelming evidence that whenever drugs, including the unsaturated oils, were used to lower serum cholesterol, mortality increased, from a variety of causes including accidents, but mainly from cancer.
Since the l930s, it has been clearly established that suppression of the hyroid raises serum cholesterol (while increasing mortality from infections, cancer, and heart disease), while restoring the thyroid hormone brings cholesterol down to normal.

In this situation, however, thyroid isn’t suppressing the synthesis of cholesterol, but rather is promoting its use to form hormones and bile salts. When the thyroid is functioning properly, the amount of cholesterol in the blood entering the ovary governs the amount of progesterone being produced by the ovary, and the same situation exists in all steroid-forming tissues, such as the adrenal glands and the brain.

Progesterone and its precursor, pregnenolone, have a generalized protective function: antioxidant, anti-seizure,
antitoxin, anti-spasm, anti-clot, anticancer, pro-memory, pro-myelination, pro-attention, etc. Any interference with the
formation of cholesterol will interfere with all of these exceedingly important protective functions.
As far as the evidence goes, it suggests that coconut oil, added regularly to a balanced diet, lowers cholesterol to
normal by promoting its conversion into pregnenolone.

Coconut-eating cultures in the tropics have consistently lower cholesterol than people in the U.S. Everyone that I know
who uses coconut oil regularly happens to have cholesterol levels of about 160, while eating mainly cholesterol rich
foods (eggs, milk, cheese, meat, shellfish). I encourage people to eat sweet fruits, rather than starches, if they want
to increase their production of cholesterol, since fructose has that effect.

Many people see coconut oil in its hard, white state, and — as a result of their training watching television or going
to medical school — associate it with the cholesterol-rich plaques in blood vessels. Those lesions in blood vessels are
caused mostly by lipid oxidation of unsaturated fats, and relate to stress, because adrenaline liberates fats from
storage, and the lining of blood vessels is exposed to high concentrations of the blood-borne material.

In the body, incidentally, the oil can’t exist as a solid, since it liquefies at 76 degrees. (Incidentally, the
viscosity of complex materials isn’t a simple matter of averaging the viscosity of its component materials; cholesterol
and saturated fats sometimes lower the viscosity of cell components.)
Most of the images and metaphors relating to coconut oil and cholesterol that circulate in our culture are false and
misleading. I offer a counter-image, which is metaphorical, but it is true in that it relates to lipid oxidation, which
is profoundly important in our bodies. After a bottle of safflower oil has been opened a few times, a few drops that get
smeared onto the outside of the bottle begin to get very sticky, and hard to wash off.

This property is why it is a valued base for paints and varnishes, but this varnish is chemically closely related to the
age pigment that forms “liver spots” on the skin, and similar lesions in the brain, heart, blood vessels, lenses of the
eyes, etc. The image of “hard, white saturated coconut oil” isn’t relevant to the oil’s biological action, but the image
of “sticky varnish-like easily oxidized unsaturated seed oils” is highly relevant to their toxicity.

The ability of some of the medium chain saturated fatty acids in coconut oil to inhibit the liver’s formation of fat
very likely synergizes with the pro-thyroid effect, in allowing energy to be used, rather than stored.
When fat isn’t formed from carbohydrate, the sugar is available for use, or for storage as glycogen. Therefore, shifting
from unsaturated fats in foods to coconut oil involves several anti-stress processes, reducing our need for the adrenal
hormones. Decreased blood sugar is a basic signal for the release of adrenal hormones.
Unsaturated oil tends to lower the blood sugar in at least three basic ways.

It damages mitochondria, causing respiration to be uncoupled from energy production, meaning that fuel is burned without
useful effect. It suppresses the activity of the respiratory enzyme (directly, and through its anti-thyroid actions),
decreasing the respiratory production of energy.
And it tends to direct carbohydrate into fat production, making both stress and obesity more probable. For those of us
who use coconut oil consistently, one of the most noticeable changes is the ability to go for several hours without
eating, and to feel hungry without having symptoms of hypoglycemia.

One of the stylish ways to promote the use of unsaturated oils is to refer to their presence in “cell membranes,” and to
claim that they are essential for maintaining “membrane fluidity.” As I have mentioned above, it is the ability of the
unsaturated fats, and their breakdown products, to interfere with enzymes and transport proteins, which accounts for
many of their toxic effects, so they definitely don’t just harmlessly form “membranes.”
They probably bind to all proteins, and disrupt some of them, but for some reason their affinity for proteolytic and
respiration-related enzymes is particularly obvious. (I think the chemistry of this association is going to give us some
important insights into the nature of organisms).

Unsaturated fats are slightly more water-soluble than fully saturated fats, and so they do have a greater tendency to
concentrate at interfaces between water and fats or proteins, but there are relatively few places where these interfaces
can be usefully and harmlessly occupied by unsaturated fats, and at a certain point, an excess becomes harmful.
We don’t want “membranes” forming where there shouldn’t be membranes. The fluidity or viscosity of cell surfaces is an
extremely complex subject, and the degree of viscosity has to be appropriate for the function of the cell.
Interestingly, in some cells, such as the cells that line the air sacs of the lungs, cholesterol and one of the
saturated fatty acids found in coconut oil can increase the fluidity of the cell surface.
In red blood cells, which have sometimes been wrongly described as “hemoglobin enclosed in a cell membrane,” it has been
known for a long time that lipid oxidation of unsaturated fats weakens the cellular structure, causing the cells to be
destroyed prematurely.

Lipid oxidation products lower the rigidity of regions of cells considered to be membranes. But the red blood cell is
actually more like a sponge in structure, consisting of a “skeleton” of proteins, which (if not damaged by oxidation)
can hold its shape, even when the hemoglobin has been removed. Oxidants damage the protein structure, and it is this
structural damage which in turn increases the “fluidity” of the associated fats.

So, it is probably true that in many cases the liquid unsaturated oils do increase “membrane fluidity,” but it is now
clear that in at least some of those cases the “fluidity” corresponds to the chaos of a damaged cell protein structure.
(N. V. Gorbunov, “Effect of structural modification of membrane proteins on lipid-protein interactions in the human
erythrocyte membrane,” Bull. Exp. Biol. & Med. 116(11), 1364-67. 1993.

Although I had stopped using the unsaturated seed oils years ago, and supposed that I wasn’t heavily saturated with
toxic unsaturated fat, when I first used coconut oil I saw an immediate response, that convinced me my metabolism was
chronically inhibited by something that was easily alleviated by “dilution” or molecular competition.
I had put a tablespoonful of coconut oil on some rice I had for supper, and half an hour later while I was reading, I
noticed I was breathing more deeply than normal. I saw that my skin was pink, and I found that my pulse was faster than
normal — about 98, I think. After an hour or two, my pulse and breathing returned to normal.

Every day for a couple of weeks I noticed the same response while I was digesting a small amount of coconut oil, but
gradually it didn’t happen any more, and I increased my daily consumption of the oil to about an ounce. I kept eating
the same foods as before, except that I added about 200 or 250 calories per day as coconut oil.

Apparently the metabolic surges that happened at first were an indication that my body was compensating for an anti-
thyroid substance by producing more thyroid hormone; when the coconut oil relieved the inhibition, I experienced a
moment of slight hyperthyroidism, but after a time the inhibitor became less effective, and my body adjusted by
producing slightly less thyroid hormone.

But over the next few months, I saw that my weight was slowly and consistently decreasing. It had been steady at 185
pounds for 25 years, but over a period of six months it dropped to about 175 pounds. I found that eating more coconut
oil lowered my weight another few pounds, and eating less caused it to increase.

The anti-obesity effect of coconut oil is clear in all of the animal studies, and in my friends who eat it regularly.
It is now hard to get it in health food stores, since Hain stopped selling it. The Spectrum product looks and feels a
little different to me, and I suppose the particular type of tree, region, and method of preparation can account for
variations in the consistency and composition of the product.

The unmodified natural oil is called “76 degree melt,” since that is its natural melting temperature. One bottle from a
health food store was labeled “natural coconut oil, 92% unsaturated oil,” and it had the greasy consistency of old lard.
I suspect that someone had confused palm oil (or something worse) with coconut oil, because it should be about 96%
saturated fatty acids.

Raymond Peat, Ph.D.
P.O. Box 5764
Eugene, OR 97405

THE REMARKABLE BENEFITS OF COCONUT OIL
2

Coconut Oil: The Healthiest Oil on Earth
By Brian Shilhavy

“Coconut oil is the healthiest oil on earth,” says Dr. Bruce Fife, a naturopathic doctor and the author of the book The
Healing Miracles of Coconut Oil. Modern research seems to back up this bold statement. Once wrongly accused of
increasing cholesterol levels, coconut oil is now actually being used by doctors in the treatment of a variety of
disorders. Clinical studies have shown that coconut oil has anti-microbial and anti-viral properties, and is now even
being used in treating AIDS patients. Studies conducted in the Philippines last year showed that coconut oil does indeed
reduce the viral load in AIDS patients.

Lauric Acid: A Key Component to Health

Lauric acid is a medium chain fatty acid which is abundant in coconut oil and is considered responsible for many of its
health benefits. Coconut oil is about 50 percent lauric acid. The only other abundant source found in nature is in human
breast milk.

Dr. Jon J. Kabara, PhD. and Professor Emeritus of Michigan State University, says, “Never before in the history of man is it so important to emphasize the value of lauric oils. The medium-chain fats in coconut oil are similar to fats in mother’s milk and have similar nutriceutical effects.”
Dr. Mary Enig, a nutritionist/biochemist and one of the world’s leading authorities on fats and oils, goes on to say, Approximately 50 percent of the fatty acids in coconut fat are lauric acid. Lauric acid is a medium chain fatty acid, which has the additional beneficial function of being formed into monolaurin in the human or animal body. Monolaurin is the antiviral, antibacterial, and antiprotozoal monoglyceride used by the human or animal to destroy lipid coated viruses such as HIV, herpes, cytomegalovirus, influenza, various pathogenic bacteria including listeria monocytogenes and heliobacter pylori, and protozoa such as giardia lamblia. Some studies have also shown some antimicrobial effects of
the free lauric acid.”

The Politics of Tropical Oils

So why has coconut oil gotten such a bad rap in the recent past? After all, much of the research supporting coconut oil as a healthy fat has been around for some time. The answer is politics and economics. Coconut oil was heavily used in the U.S. at one time, being used for baking, pastries, frying, and theater popcorn. But starting in the 1980s, some very powerful groups in the U.S. including the American Soybean Association (ASA), the Corn Products Company (CPC
International), and the Center for Science in the Public Interest (CSPI) began to categorically condemn all saturated oils. Faulty science was used to convince the public that ALL saturated fats were unhealthy, when in fact saturated fats rich in the medium-chain fatty acids like lauric acid are very healthy.

These organizations were are aided by the United States Food and Drug Administration (FDA), many of whose key personnel are recruited from and return to the vegetable oil industry. The result was that most people switched to vegetable oils, and the main source of lauric acid from tropical oils in the American diet was lost. The countries that these tropical oils came from, mainly the Philippines and Malaysia, were too poor to counter these untrue claims with advertising
investments for the truth. It is only recently that the health benefits of these tropical oils are starting to become rediscovered.

Population Studies

While some clinical studies have been conducted recently, such as the study on AIDS patients in the Philippines (1999 –
2000), much of the studies have been done on tropical populations where coconut products are a main part of the diet.

One such study was done in the South Pacific islands of Pukapuka and Tokelau near New Zealand. The studies were started
in the 1960s before either island was exposed to Western refined food. These populations ate only natural foods, and
coconut foods were the most prevalent, being consumed at each meal in one form or another. While most people in western
countries get 30-40 percent of their calories from fats, the people in these islands averaged between 50 and 60 percent
of their calories from fat, most of that being saturated fat from coconuts.

So what kind of health did these studies find among the populations in these two islands? Bruce Fife reports in his
book: “The overall health of both groups was extremely good compared to Western standards. There were no signs of kidney
disease or hypothyroidism that might influence fat levels. There was no hypercholesterolemia (high blood cholesterol).
All inhabitants were lean and healthy despite a very high saturated-fat diet. In fact, the populations as a whole had
deal weight-to-height ratios as compared to the Body Mass Index figures used by nutritionists. Digestive problems are
rare. Constipation is uncommon. They average two or more bowel movements a day. Atherosclerosis, heart disease, colitis,
colon cancer, haemorrhoids ulcers, diverticulosis, and appendicitis are conditions with which they are generally
unfamiliar.”

A fat that causes weight loss?

Another incredible fact about coconut oil is that even though it is a fat, it actually promotes weight loss! The reason
is again because of the healthy medium-chain fatty acids. These fatty acids do not circulate in the bloodstream like
other fats, but are sent directly to the liver where they are immediately converted into energy, just like
carbohydrates. So the body uses the fat in coconut oil to produce energy, rather than be stored as body fat. Medium-
chain fatty acids found in coconut oil also speed up the body’s metabolism burning more calories and promoting weight
loss. The weight loss effects of coconut oil have clearly been demonstrated by many researchers. (A list of references
can be found in Bruce Fife’s book The Healing Miracles of Coconut Oil.)

Therapeutic Dosage

So how much coconut oil should one consume? A good therapeutic dosage is 3 to 4 tablespoons a day. This provides enough
lauric acid to build the immune system. Also, look for unrefined coconut oil. Stay away from all hydrogenated oils,
whether it is coconut oil or vegetable oils. Hydrogenated oils are oils with trans-fatty acids, which have been altered
from their original chemical composition and have been shown to raise serum cholesterol levels that can lead to heart
disease.

Also look for unrefined coconut oils like Virgin Coconut Oil. Most commercial coconut oils are RBD (refined, bleached,
and deodorized). While these RBD oils do maintain the beneficial chemical structures of the medium chain fatty acids,
they also contain chemicals used in processing.

Brian Shilhavy is a Certified Nutritional Counselor (CNC).

from: http://www.naturodoc.com/
Read: Part One of the Benefits of Coconut Oil

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